The lachrymation, conjunctival injection, and nasal moisture are transient symptoms which are usually absent after the first day. In this connection it is interesting to note that Mr. Jonathan Hutchinson, as early as 1866, recognized many of the ocular symptoms of paralysis of the cervical sympathetic, and wrote a paper thereon. HOW DOES EXCISION OF THE CERVICAL SYMPATHETIC REDUCE INTRA-OCULAR TENSION? This is a question difficult to answer—difficult for the reason that we are not sufficiently acquainted with the physiology of the production of aqueous humor under normal surroundings. Panas and Duvigneaud have assumed rightfully that “If the nervous mechanism of intra-ocular secretion or, to speak without hypothesis, the action of the nervous system on intra-ocular tension can be known, the pathology of glaucoma will be cleared up, iridectomy will be explained, and perhaps a new and scientific basis for the treatment of glaucoma will be established.” Many observers have sought to solve the problem. Donders attributed the hypertension to a neuro-secretory cause and believed the trigeminus to be the agent of excessive secretion. He held that section of the trigeminus should relieve intra-ocular tension, while section of the cervical sympathetic could have no particular influence. His views were overthrown by experiments made by Wegner in 1866, on rabbits. By means of manometers placed in the anterior chamber, he sought to record variations in the intraocular tension. He proved to his own satisfaction that the trigeminus takes no part, while section of the cervical sympathetic produces hypotonia, and irritation of its upper end and causes hypertonia. He held that section of the cervical sympathetic enlarges the blood vessels of the eye; the blood then flows under reduced pressure, and intra-ocular secretion is lessened. Almost identical results were obtained by Adamück—1866‒68—who experimented on cats. Von Hippel and Gruenhagen believed that the cervical sympathetic contains vasoconstrictor fibers for the eye. Their experiments were made on cats and dogs. They found that irritation of the upper end of the cervical sympathetic causes in the cat hypertonia, while its extirpation increases intra-ocular tension. While, according to Wegner, the hypertonic action proceeds from the enlargement of vessels caused by cutting the cervical sympathetic, and the contraction of the blood vessels caused by the irritation of the nerve causes a hypertonic action, the contrary view is held by Adamück, Von Hippel, and Gruenhagen. However this may be, there is no doubt that the trigeminus plays no great part in the production of ocular tension. Furthermore, the inefficiency of Bedal’s operation—stretching the nasal nerve—is explained by the fact that it is the cervical sympathetic, and not the trigeminus, which influences intra-ocular tension.