A History of Epidemics in Britain, Volume 1 (of 2): From A.D. 664 to the Extinction of PlagueCreighton, Charles
History
A History of Epidemics in Britain, Volume 1 (of 2): From A.D. 664 to the Extinction of Plague
Creighton, Charles
Epidemics -- Great Britain -- History
Let us add to this, that the meat diet of the poorer class, whether serfs
or freemen, would be apt to consist of the more worthless portions, the
semi-putrid pieces in the salted sides of bacon, mutton or beef, and that
badly-cured pork was in many parts the usual kind of flesh-food; and we
shall have no difficulty in finding the noxious element in the diet of the
Middle Ages, which the dietetic hypothesis of leprosy requires. Some who
have advocated that hypothesis for modern leprosy, have laid themselves
open, notwithstanding the ability and industry of their research, to
plausible objections which have no bearing if the hypothesis be
sufficiently safe-guarded. Leprosy, like every other _morbus miseriae_,
needs a number of things working together to produce it, its more or less
uniform specific character or distinctive mark being determined by the
presence of one factor in particular. The special factor should be
generalised as much as possible, so as to cover the whole circumstances of
leprosy: it is not only half-cured or semi-putrid fish[208], but
half-cured or semi-putrid flesh of any kind. The most general expression
for leprosy is a semi-putrid or toxic character of animal food, just as
for the allied pellagra, it is a semi-putrid or toxic character of the
bread or porridge. Moreover it is that noxious or unnatural thing in the
food, not once and again, or as a _bonne bouche_, but somewhat steadily
from day to day as a chief part of the sustenance, and from year to year.
As the rain-drops wear the stones, so the poison in the daily diet tells
upon the constitution. Once more, such special causes may be present in a
country generally, among the poor of all the towns, villages and hamlets,
and yet only one person here and there may show specific effects that are
recognisable as a disease to which we give a name. Unless there be present
the aiding and abetting things, the special factor will hardly make itself
felt; and if there be not the special factor, there may be some other
_morbus miseriae_ but there will not be that one. These aiding things are
for the most part the usual concomitants of poverty and hardships, wearing
out the nerves far more than is commonly supposed and producing in
ordinary an excessive amount of nervous affections among the poor. But
among the poor themselves, as well as among the well-to-do, there are
special susceptibilities in individuals and in families. One person may
have the same unwholesome surroundings as another and the same poisonous
element in his diet, but he may fall into no such train of symptoms as his
leprous neighbour because he is not formed in quite the same way, because
he has “no nerves,” or is of a hardier stock, or because his unwholesome
manner of life comes out in some other form of disease (scrofula perhaps,
less probably gout), or for some other reason deeply hidden in his
ancestry and his personal peculiarities. The chances would be always
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