A Text-Book of Horseshoeing, for Horseshoers and VeterinariansLungwitz, A. (Anton)
Science
A Text-Book of Horseshoeing, for Horseshoers and Veterinarians
Lungwitz, A. (Anton)
Horseshoeing
The seat of the disease is in the fleshy leaves about the toe, more
rarely upon the side walls and quarters. Depending upon the intensity
of the inflammation, the fleshy leaves are more or less loosened
from the horny leaves, as a result of which there is a change of
position of the os pedis, with a simultaneous sinking of the coronet
at the toe. This produces a change of form of the hoof. The quarters
become higher. Rings form upon the wall, and their course is quite
characteristic of the disease. At the toe these rings are quite close
to one another, but as they pass back towards the quarters they
gradually separate from one another and recede from the coronary band
(Figs. 193, 194, and 195).
[Illustration: FIG. 193.
Vertical longitudinal section of a foot altered by chronic laminitis:
_a_, hollow wall at toe thrust forward; _b_, leafy layer much thickened
and crumbling (“seedy-toe”); _c_, dotted line showing limit to which
the toe may be rasped away in shoeing; _d_, dropped sole; _e_, atrophy
of lower sharp edge of os pedis; _g_, dotted line indicating the height
of the perioplic band; _h_, foot axis.]
The wall at the toe is sunken just under the coronet; its lower part,
on the contrary, is thrust forward. _Later, the white line becomes
pathologically widened._ The horn of the white line is dry, crumbling,
and easily broken down, so that a break in continuity (crack) is apt to
occur between the wall and sole, and lead to the formation of a _hollow
wall_ (“seedy-toe”). Where the inflammation is moderate and is not
repeated, healing usually takes place and the horn grows down regularly
and in normal direction from the coronet. However, a rather brittle
condition of the horn remains permanently. If, on the contrary, the
inflammation was very severe or repeated several times, the horny sole
becomes flat just in front of the point of the frog as a result of the
sinking of the os pedis, or it may even drop below the level of the
wall (full hoof, dropped sole). Indeed, it even happens at times that
the toe of the os pedis perforates the horny sole just in front of the
point of the frog. The wall at the toe, which was previously but little
altered in form, is now thrust prominently forward.
[Illustration: FIG. 194. Foundered foot (chronic laminitis), before
dressing.]
[Illustration: FIG. 195.
Foundered foot, dressed and shod. The dotted lines indicate its form
before being dressed,—_i.e._, as shown in Fig. 194.]
The inflammation of the pododerm may under certain conditions and by
skilful veterinary treatment be removed, so that the characteristic
changes of form and quality of the hoof will not occur. But if this is
not accomplished, as is often the case, the disease will be obstinate,
and permanent morbid changes of the horn capsule take place.
[Illustration: FIG. 196.
Public-domain text, read in full here on John Shaqi.
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