The =symptoms= are characterized by acute gastro-intestinal disturbances. The onset is usually sudden. The first to appear are severe griping pains in the abdominal region, there may be various nervous manifestations, such as drowsiness, muscular twitchings and more or less restlessness. The abdominal pains may be accompanied with diarrhœa, nausea and vomiting. There may be chills and headache. As the condition progresses the stools become a greenish color and of a very watery consistency. There may be chills and headaches, marked muscular weakness, faintness and possibly prostration. The temperature runs from 102° F. to 103° F. There is excessive thirst, skin eruptions and herpes. There is often oliguria. The severity of the symptoms will depend upon the amount of poison taken into the system and the freedom of transmission of mental impulses, which will enable Innate Intelligence to bring about her adaptative processes. Usually the attack lasts only a few days, although the fulminating cases may prove fatal within twenty-four hours. Warm weather seems to be conducive to the formation of this poison, since the greater number of outbreaks occur in the summer time. Food Intoxication This form of food poisoning is called botulism. The poison which forms is the specific toxin produced by the activity of the bacillus botulinus. The botulis itself lives on decayed organic matter, therefore it is of the saprophyte type. This poison is found in a great many different foods of both plant and animal origin. The intoxication in botulism affects the central nervous system. It is a febrile condition and there are no gastro-intestinal disturbances. Usually the symptoms in botulism will appear from eighteen to thirty-six hours after the poison has been taken into the stomach. There are cases on record in which the symptoms have appeared within four hours. The period of incubation depends upon the amount of toxin ingested and the ability of Innate Intelligence to adapt the body to the poison. The =symptoms= usually begin with headache and dizziness, feeling of fatigue and muscular weakness. One of the early symptoms is a disturbance of vision which may progress until the patient is blind. Both the extrinsic and intrinsic muscles of the eye become involved. There is blepharoptosis and the pupils become dilated. There is diplopia and the loss of adaptative response of the eye to the light. There is soon complete loss of accommodation, opthalmoplegia, nystagmus, strabismus and in some cases photophobia.