Disease in captive wild mammals and birds : $b incidence, description, comparisonFox, Herbert
Science
Disease in captive wild mammals and birds : $b incidence, description, comparison
Fox, Herbert
Veterinary medicine
To what extent do the parasites predispose to pneumonia? Compare for
this purpose the two columns of Table 8 showing active and inactive
pulmonary parasitism. In Rodentia and Galli alone do we note that active
parasitism is effective, there being no passive cases. In all the other
orders, animal invaders of the lung are more often encountered as
quiescent or encapsulated bodies, therefore as findings incidental to
the autopsy and perhaps not concerned in the cause of death. In Ungulata
the inactive parasitisms are five times as frequent as the active
inflammatory lesions. It might be added that the list is made up of
cases wherein we saw parasites whether determined or not, an explanation
of the apparently small number of cases; there were many more in which
such invaders were suspected but not found and therefore excluded.
BRONCHIECTASIS.
Bronchiectasis affecting the smallest tubes, or bronchiolectasis, is not
at all uncommon in verminous pneumonitis and is explained as due to the
degeneration of the wall, the surrounding progressive ulceration, to
accumulation of inspired air and its retention by the obstruction. There
is described a generalized bronchiolectasis, chiefly in young human
beings, due to a destructive bronchiolitis; this has not been seen.
Non-verminous bronchiectasis of the middle sized bronchi such as is seen
in human chronic bronchitis, simple or tuberculous, is quite uncommon.
Widening of the bronchial lumen may be divided, as I see its
pathogenesis, into (a) that due to congenital weakness of the walls, (b)
that due to obstruction permitting air to pass into but not out of a
bronchus because of a ball-valve obstruction or weakness of expiratory
power, (c) that due to external pressure by tumors or distortion by
fibrous tissue either within the lung or pleura and (d) that due to
inflammatory weakening of walls, augmented by loss of supporting
pulmonary tension, accummulation of secretion and the dilating effect of
inspiration preparatory to and incidental to coughing. How important the
last three auxiliary factors may be in the cases explicable under a, b,
c, can easily be speculated upon and may vary in different cases.
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