TABLE 13. _Showing the Incidence of Gastroenteric Disease in the Two Forms of Marsupial Intestinal Tracts._ ═══════════════════════╤═══════════════════════╤═══════════════════════ │ Group A │ Group B │Carnivorous Stomach and│Herbivorous Stomach and │ Intestines 103 │Intestines 73 Specimens │ Specimens Per cent. │ Per cent. ───────────────────────┼───────────────────────┼─────────────────────── Acute gastritis │ 9.6 │ 15. │ Bacteria Verminous │ Bacteria Verminous Chronic gastritis │ 0. 13.6 │ 5.4 0. Acute enteritis │ 2.9 │ 1.5 Colitis incl. typhlitis│ 1.9 │ 4.3 ───────────────────────┴───────────────────────┴─────────────────────── Table 13 contains an analysis of the forms of gastroenteritis as they were described in the two groups of tracts, that resembling the carnivorous, that similar to the herbivorous intestinal construction, and according to the factor believed to be responsible. In the first group gastritis of an acute nature occurred in 9.6 per cent. of the 103 specimens. They were chiefly catarrhal in character and seem for the most part secondary to verminous infestation; at least six of the ten cases were associated with parasites. The process microscopically is catarrhal and deeply infiltrative. Group B has a high incidence of gastritis and here the evidence of bacteria or toxins is quite plain. Several of the cases were in animals showing also Kangaroo disease of the jaw with pneumonia or septicemia; the gastric lesion of streptothricosis will be described under that heading. The character of gastritis without jaw disease is somewhat different from that with it. Pathologically the process is a congestive and superficially necrotizing affair, forming upon the tips of the folds, small gray erosions or flat shallow irregular ulcers, which upon histological study consist of loss of tissue of the mucosa and some deep congestion with round cell groups but no reaction deep in mucosa or submucosa. True catarrhal inflammation has occurred, but not like in the opossums.