At first the bowels are constipated, but in time fœtid diarrhœa sets in. Examination of the digestive apparatus on the left side and in the right posterior abdominal region reveals nothing abnormal, but pressure over the lower portion of the abdomen and along the cartilages of the right hypochondriac region produces, on the contrary, well-marked pain. This region corresponds to the position of the abomasum. The conjunctiva appears reddish yellow, as in most visceral inflammations. Some authors have described attacks of extreme excitement, but these are no more pathognomonic than is grinding of the teeth, which is a constant symptom, or the metallic sound noted on auscultation of the rumen. This sound occurs in all cases of inertia of the rumen, and indicates distension and emptiness of the viscus (acute peritonitis, chronic adhesive peritonitis, inflammation of the reticulum as a consequence of the presence of foreign bodies). Dull colic and groaning are not uncommon. Finally, Thierry and others have all mentioned an alliaceous smell of the eructations as pathognomonic. Acute gastritis develops regularly in ten to fifteen days, after which the symptoms diminish and disappear, giving place to normal health. In grave cases, despite proper treatment, acute gastritis more frequently ends in a chronic condition, finally leading to gastric atrophy, and the insufficient secretion of hydrochloric acid, with all the consequences of these conditions. The glands of the stomach degenerate; the secretion becomes abnormal and dyspepsia is set up. =Diagnosis.= The diagnosis is rather difficult, for the condition is very apt to be confused with primary dyspepsia, or with inflammation of the gastric compartments. It might also be mistaken for acute enteritis of the first part of the small intestine; but as gastritis is very often complicated with duodenitis, such a mistake is without serious consequences. =Prognosis.= The prognosis is grave, not because death is a frequent termination, but because the disease very often leads to chronic incurable lesions. =The lesions= consist of congestion of the vascular network of the mucous and sub-epithelial coats, serous infiltration of the corium and submucous connective layers, desquamation, and later in excessive proliferation of the epithelium. When the inflammation is deep seated the epithelium of the gastric glands becomes swollen and cloudy, and undergoes a kind of atrophic degeneration. In very grave cases, petechiæ, superficial capillary hæmorrhages, and slight ulceration may be noted. The mucous folds are always thickened and infiltrated.