On the right side, on the contrary, palpation causes the animal to resist and to show signs of pain. According as this sensitiveness is more marked in the middle or upper region or towards the hypochondriac circle, we infer that the inflammation is most acute in the large or small intestine, either in the middle portions or, again, in the most anterior portion. The temperature always rises at the commencement, attaining 103° to 104° Fahr. (39·5° or 40° C.), but rarely a higher point, a fact which negatives the idea of a rapidly progressive infectious disease. Slight colic appears, and is accompanied by constipation; the fæces are covered with mucus, or false membranes, or are completely enclosed in fibrinous tubes. After four or five days the fæces change in character. Constipation gives place to a liquid, blackish, very fœtid diarrhœa. Finally the mouth exhales a stercoraceous odour, resulting from fœtid eructation. The passage of flat or tubular false membranes continues for a certain time. When false membranes constitute the chief symptom, the condition is termed “pseudo-membranous, croupal, or diphtheritic enteritis.” This form is only a variety of acute enteritis, but is grave, because complications due to hæmorrhage or infection are very liable to occur. In certain cases movement causes groaning. In the slighter forms the patient may recover spontaneously. If fed with easily digested materials, the symptoms diminish in intensity. Towards the eighth day the fæces become normal, appetite and rumination reappear, and the secretion of milk which had fallen off rises to its normal amount. Resolution has occurred. More frequently, either because the animals have less power of resistance or because decomposition is taking place more actively in the intestine, or, again, because intoxication is setting in, the disease becomes aggravated. Constipation is more marked, and the fæces passed are in small masses, covered with layers of epithelium, or sometimes streaked with blood. In other cases the diarrhœa becomes exaggerated and assumes a mucous or sero-mucous character; the temperature rises, and death results from exhaustion and infection, microorganisms passing from the lumen of the intestine through its walls and invading the general circulation. =Lesions.= To study the lesions to advantage the autopsy must be performed as soon as possible after death. In slight cases they consist of trifling generalised congestion of the intestinal mucous membrane. The points most affected show infiltration of the submucous coat, and sometimes of the muscular coat; the wall of the intestine is double its normal thickness, but the thickening never equals that seen in cases of rapidly fatal intestinal congestion.