Diseases of the Horse's FootReeks, H. Caulton (Harry Caulton)
Science
Diseases of the Horse's Foot
Reeks, H. Caulton (Harry Caulton)
Hoofs -- Diseases; Horses -- Diseases
In the case of a deep but recent crack there is always more or less
hæmorrhage. This favours risk of infection of the lesion with pus-forming
organisms, and so leads to a more or less pronounced lameness, a degree of
swelling, heat and tenderness in the coronet above, and a certain amount of
surgical fever.
The acute symptoms subdued, but the fissure still remaining, gives us the
crack we have classified as 'old.' This may in every case be distinguished
from a more recent lesion by the amount of thickening of the overhanging
coronet, and the presence of an increased quantity of sub-coronary horn in
the region immediately about the crack. The previous inflammatory changes
in the adjoining sensitive structures have here led to an increased
secretion of horn, and a greater or less deposition of inflammatory
connective tissue in the wounded coronary cushion.
Sand-crack of the toe always follows the direction of the horn fibres. That
of the quarter, however, may on occasion run a course that is somewhat
zigzag, first following the direction of the horn fibres for a short
distance, then travelling in a horizontal direction, and finally continuing
its course again in a line with the horn fibres, commonly at a point
posterior to that at which it commenced.
In a quarter-crack that is old, and when contraction of the heels exists
(which in this case it usually does), then will often be found overlapping
of the edges of the crack. The expansion of the wall brought about when the
body-weight is on the heels, cannot, by reason of the break in it, continue
itself anterior to the crack. As a consequence, repeated expansion of the
wall posterior to the crack, with the portions anterior to it in a state
of enforced quiescence, leads in time to the posterior edge of the crack
coming to lie over that of the anterior.
_Complications_.--The first complication likely to arise in a case of
sand-crack is that attending simple laceration of the sensitive structures
in a _deep_ lesion. With the laceration all the phenomena of a repairing
inflammation make their appearance. As a result, there is more or less heat
according to the degree of inflammatory hyperæmia, swelling according to
the amount of inflammatory exudate, and pain according to the amount of
pressure the two foregoing bring to bear on the nerves in the inflamed
area.
A second and more serious complication is the greater inflammation set up
by the introduction into the crack of foreign substances. Small portions of
gravel and flint, both by the irritation set up by their friction and by
the infection they carry in with the dirt surrounding them, are responsible
for the mischief.
When, from direct communication with the blood-stream, due to extensive
hæmorrhage, bacteria from the outside gain entrance, this simple
inflammation is further complicated by the formation of pus, or a limited
gangrene of the keratogenous membrane.
Public-domain text, read in full here on John Shaqi.
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