The artery that ruptures in the brain, in cases of apoplexy, {146} is practically always the same. Its scientific name is the lenticulo-striate artery, but it is oftener called by the name given it by Charcot--the artery of cerebral hemorrhage. The reason why arteries in the brain rupture rather than arteries in other organs is that in the brain, in order to avoid the demoralising effect of too sudden changes of blood pressure upon the nervous substance, the cerebral arteries are terminal, are not connected directly with a network of finer arteries as in the rest of the body, but gradually become smaller and smaller, and end in the capillary network which is the beginning of the venous vascular system. This special artery ruptures, because it is almost on a direct line from the heart, and so blood pressure is higher in it than in other brain arteries. The tradition that people with short necks are a little more liable to apoplexy than are those of longer cervical development has a certain amount of truth in it, though not near so much as is often claimed for it. Another predisposing element to apoplexy is undoubtedly heredity. Families have been traced in which, for five successive generations, there have been attacks of apoplexy between fifty-five and sixty years of age. Short-necked people, with any history of apoplexy in the family, should especially be careful, if they have any of the symptoms--dizziness, sleepy fingers, etc.--that we have already noted. There is a tradition that the third stroke of apoplexy is always fatal. This is without foundation in experience, though of course the liability of death increases with each stroke, and few patients survive the third attack. I remember seeing in Mendel's clinic, in Berlin, a man who was suffering from his seventh stroke and promised to recover to have another. Each successive stroke is much more dangerous to life than the preceding one, however. In general, the prognosis of an apoplexy, that is to say what the ultimate result will be, is impossible. The patient may come to in an hour or two, and may not come out of the coma at all. There is no way of deciding how large the artery is that is ruptured, nor how much blood has been effused into the brain, nor how much damage has been done to important nerve centres. Nor is there any {147} effective way of stopping the effusion, though certain things seem to be of some benefit in this matter. We can only wait, assured that, in most of the cases, the patient will have a return of consciousness, at least for a time.