Gout, with a section on ocular disease in the goutyLlewellyn, Llewellyn J. (Llewellyn Jones)
Science
Gout, with a section on ocular disease in the gouty
Llewellyn, Llewellyn J. (Llewellyn Jones)
Eye -- Diseases; Gout
_Plantar Neuralgia._—Occasionally, as we have pointed out elsewhere,
the pain is of almost unendurable severity. It constitutes one of the
types of so-called _partial sciatica_, the pain and paræsthesia being
confined to the plantar nerves. Indeed, pain, numbness, hyperæsthesia,
or sweating of the sole are often symptomatic of a definite neuritis.
Such may follow typhoid fever or caisson disease, and in this latter be
of aggravated type. When we realise that the pain in these cases may be
limited to the tips of the toes or the _ball of the great toe_, we see
how readily it may be confused with “gout.” Fortunately plantar neuralgia
is exceptionally rare; but even after exclusion of the foregoing causes
we should, before pronouncing any such neuralgia to be “gouty,” recollect
that plantar neuralgia or hyperæsthesia is very common in _alcoholism_
and _hysteria_.
_Erythromelalgia._—Among the exceptional cases that find their way to
spas are examples of this rare disorder. Almost invariably they come
under the diagnosis of “gout” or “rheumatism.” When we reflect that in
the majority of instances the initial burning pain typical of the disease
is located in some part of the sole of one foot, and that the associated
redness and vascularity may be delimited to the _ball of the great toe_,
the heel, or outer or inner side of the foot, we see the danger of its
being too easily referred to “gout.”
If seen at the zenith of an attack, the severe pain, the local heat,
the intense purplish redness, the distension of the veins, and in some
instances œdema, how close the resemblance to gout! Precisely also, as
in gout, the simulation of a deep-seated inflammatory process is very
pronounced. Indeed, in not a few examples of erythromelalgia fruitless
incisions have been made. Accordingly in all cases of pain, redness, and
swelling in the sole of the foot, we should canvass the possibility that
we may be face to face with an instance of erythromelalgia, a disorder
which, like gout, is most frequently met with in men of middle age.
In drawing to a close our remarks on the diagnosis of _acute gout_ in
the _foot_, we would emphasise the fact that in all such cases the
bare feet should be thoroughly examined. For, apart from _infective_
and _traumatic_ lesions, the frequency with which the various _static
foot deformities_ are confused with “gout” is incredibly common. That
_gout may co-exist with hallux valgus_ or other distortion we readily
admit, but this does not absolve us of our responsibility—correction of
the static deformity. Combine this, if you will, with constitutional
treatment if there be evidence, _i.e._, tophi, of a “gouty” diathesis,
but, we repeat, correct the mechanical defect. For gout may come and go,
but static errors remain. In so doing, the victim will be saved much
preventable suffering, and, for aught known to the contrary, the removal
of irritation and local congestion may haply minimise the chances of
subsequent gouty outbreaks.
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