Anatomy.--The theca through which the deep digital flexor (perforans) plays in the tarsal region, begins about three inches above the inner tibial malleolus and extends about one-fourth of the way down the metatarsus. The posterior part of the capsular ligament of the hock joint is very thick in its most dependent portions and is in part cartilaginous, forming a suitable groove for the passage of the deep flexor tendon. [Illustration: Fig. 57--Thoroughpin. Showing distension of the sheath of the deep flexor tendon as it protrudes antero-externally to the fibular tarsal bone (calcaneum).] Etiology and Occurrence.--Strains and sequellae to debilitating diseases constitute the usual causes of this affection. As a result of acute synovitis a chronic synovial distension of the tarsal sheath occurs. Bog spavin is often present in case of thoroughpin but the two conditions are separate and distinct excepting in that both may occur simultaneously and as the result of the same cause. Some animals are undoubtedly predisposed to disease of synovial structures. The average horse that has been subjected to hard service on pavements or hard roads at fast work suffers synovial distension of bursae, thecae or of joint capsules. Some of the well bred types such as the thoroughbred horses may be subjected to years of hard service and still remain "clean limbed" and free from all blemishes. Thus it seems that subjects of rather faulty conformation, animals having lymphatic temperaments and the coarse-bred types, are prone to synovial disturbances such as thoroughpin, bog spavin, etc., sometimes having both legs affected. [Illustration: Fig. 58--Fibrosity of tarsus as a complication in chronic thoroughpin.] Symptomatology.--Thoroughpin is characterized by a distended condition of the tarsal sheath which is manifested by protrusions anterior to the tendo Achillis. However, where but moderate distension of the sheath exists, there is little, if any, bulging on the mesial side of the hock and but a small hemispherical enlargement is presented on the outer side of the tarsus, anterior to the summit of the os calcis. In some instances the protruding parts assume large proportions, but always, because of the relationship between the fibular tarsal bone (calcaneum) and the tendon sheath, the larger protrusion is situated mesially. During the acute inflammatory stage there is marked lameness present but this soon subsides when local antiphlogistic agents are applied to the parts. In fact, spontaneous relief from lameness usually results in the course of ten days' time following the appearance of thoroughpin. No lameness marks the advent of this affection when it develops as the result of continuous strain and concussion occasioned by hard service, and local changes tend to remain in _status quo_. [Illustration: Fig. 59--Another view of same case as illustrated in Fig. 58.]