A corporal in the engineers was going the night of June 7th to a creneau of mitrailleuses, when he was bowled over by a bursting shell. He lost consciousness and was carried to the cantonment by his comrades. Next morning he complained of headache and pain in the back; had a convulsion; and proved on examination to have a left-sided hemiplegia. He was given the diagnosis of hysterical hemiplegia. He was sent to the 6th Army neurological center, and there showed a complete left-sided hemiplegia with tendency to contracture. The left knee-jerk and arm reflexes were exaggerated, and there was ankle and patella clonus with Babinski sign. There was a dysesthesia on the left side, with wrong interpretation and poor localization of painful stimuli, and non-recognition of cold and heat sensations. Muscle sense and stereognosis were impaired. There was a slight dysarthria. Lumbar puncture yielded a clear hypertensive fluid with a slight lymphocytosis. The situation remained without change for a month, when the patient was evacuated to the rear. Thus, a shell-burst can produce destructive nerve lesions without evidence of external injury. _Re_ hypertensive spinal fluid, Sollier and Chartier cite Dejerine as having brought the proof of hypertension in the cerebrospinal fluid in Shell-shock cases. They also believe that the Shell-shock hysteria is built up on a physical basis, more or less after the model of Charcot’s hysterotraumatism. Shock, windage, and gas may bring about the same kind of result. They rely especially on the cases of Sencert (201) and Ravaut (202) for their argument (1915). They recall the fact that Charcot found a hysteria due to lightning stroke and to high tension electric accidents. They quote Lermoyez as attributing like results in ear cases to labyrinthine shock, tympanic rupture, and ear hemorrhages. Shell-shock: Hemiparesis, amnesia. Lumbar punctures early (but here as late as one month after shock and after disappearance of hemiparesis) showed PLEOCYTOSIS and hyperalbuminosis. =Case 205.= (SOUQUES, MEGEVAND and DONNET, October, 1915.) A French sergeant, a machine gunner, was the victim of shell-burst September 25, 1915, was evacuated with a diagnosis of commotio cerebri, and, when examined at Paul-Brousse October 5, showed a right-sided hemiparesis, clouding of consciousness and somnolence, the hemiparesis involving the face, with deviation of tongue to right, Babinski reflex right, cremasteric and abdominal reflexes abolished on right. Normal respiration and pulse. Lumbar puncture October 7, that is, thirteen days after the injury, yielded a clear fluid with an excess of albumin, 144 small lymphocytes (some degenerate) and a single endothelial cell. October 12, the knee-jerk was a little less lively on the right side. The plantar reflex varied between extension and flexion on the right side. The cremasteric reflex had been weakly regained on the right side.