involvement, we can estimate approximately, at least, the duration of the lung condition. This makes it possible to give a relative estimate of the character of the lesions present at different periods of time. The outstanding finding, as we will discuss again, was that a distinct and peculiar pulmonary reaction was primarily imposed upon the lung, which made its appearance at periods different from those of frank lobar pneumonia. We were repeatedly surprised at finding death to have occurred during the stage of acute congestion with some hemorrhage and inflammatory œdema of lung and in the absence of any sign of grey hepatization or purulent infiltration. In many of these cases the involved areas of lung though heavy and œdematous, were still partly air-containing and the amount of lung involvement was insufficient, on the basis of mechanical interference, in accounting for the severity of the clinical symptoms and the fatal outcome. This must have impressed everyone dealing with the autopsies during the acute epidemic. It immediately suggests that in some cases at least the pulmonary lesion, in as far as incapacitating the external respiratory system, was not the sole or even the important cause of death, but that a condition of intoxication, borne out by the evidence of damage in muscles, blood and kidney is a large factor of danger in this disease. We shall briefly describe the important pulmonary findings as we have met with them in the successive stages of influenza-pneumonia. This, we hope, will make clear the interpretation of the pathology of the lung lesion of the epidemic as it came under our observation.