characteristics (and mode of deposition) appear to be quite different. It has been suggested by some that this hyaline material represents an imperfectly formed fibrin which has formed a jelly-like clot, not having the property of developing the usual threads. It is of importance to appreciate that the deposition of these hyaline structures indicates a severe injury of the alveolar walls not commonly observed in ordinary pneumonias. In different areas of the same lung these constituents of the early exudate may be observed in all proportions of admixture. Each one of the elements of the exudate may largely overshadow the others and prominently modify the appearance of the lesions. Broadly speaking, however, the inflammatory œdema and hemorrhage occupying the greatest part of the exudate in the lungs and the absence of marked leucocytic response as well as the absence of the characteristic fibrinous meshwork in the alveoli give to the early influenza-pneumonia a character different from those which we ordinarily see. It is during this early phase of the reaction that the influenza bacilli can be shown within the lung structures. The distribution of bacteria is not uniform. Clusters of these minute bacilli are found in the alveoli at irregular intervals, many of the air sacs containing much exudate being quite free from organisms. When present the bacteria appeared in tightly aggregated schools lying free amongst cells of the exudate, but also certain numbers being incorporated within the large mononuclear cells. In some regions organisms of the type of the influenza bacilli were alone seen, while elsewhere again, and particularly where the exudate was assuming purulent characters other bacteria of the nature of streptococci, staphylococci and micrococcus catarrhalis, were also found. _Lung—Secondary Stage_