The removal of the infection and the inflammatory exudate from the lung tissue is accomplished slowly. Clinically the pulmonary process clears up by lysis, and it is quite unusual to have a crisis with the rapid disappearance of the serious manifestations. It is difficult to obtain a clear conception of what takes place in any individual case recovering from an influenza-pneumonia, but if we have an understanding of what may occur in the inflamed lung tissue in any one of the stages or varieties of kind, we may visualize the changing character of the lung condition tending toward the final restoration. We have previously pointed out that the early stage of influenza-pneumonia is one of congestion, œdema, hemorrhage and more or less leucocytic infiltration, and that this reaction differs materially from that observed in pneumococcus lobar pneumonia. There being no stage of true red hepatization, it has also become apparent that this peculiar primary reaction need not pass into the stage of gray consolidation. Scattered areas in the lung pass from the condition of acute serous and hemorrhagic pneumonia to a type of purulent pneumonia while much of the remaining tissue continues in the state as seen in the early reaction. A certain amount of cellular exudate makes its appearance but not sufficient to lead to a true consolidation. This variety of reaction is present from the fifth day of the pneumonia onwards and may continue with all of its varieties through until the tenth or twelfth day or even longer when recovery from the infection is beginning. Thus the stage of resolution makes its appearance before the inflammatory reaction in the involved lobes has assumed a common character and where we are able to recognize different grades of severity and different stages of inflammation within the same lobe. Resolution taking place in such a lobe has responses occurring in the different parts determined by the nature of the antecedent reaction. We have found that those portions which have not advanced beyond the stage of œdema and hemorrhage may clear up with the disappearance of this early exudate and its infection. In a neighboring portion the purulent inflammation passes through phases differing somewhat from the preceding but also tending toward the restoration of the parenchyma and the disappearance of the inflammation. It would be incorrect to consider the resolution of the early type of inflammatory reaction as an abortive process inasmuch as it is not yet clear whether this serous and hemorrhagic process is not the characteristic inflammation of a peculiar micro-organism or organisms and that when acting alone these bacteria do not in themselves stimulate a further inflammatory response. Hence if it is true that there is a peculiar inflammatory reaction of a non-suppurative and non-fibrinous kind the manner of resolution will differ somewhat from that where these other constituents of the exudate are present. It becomes clear,