In the late purulent lesions of the lung we have encountered dilated lymphatic channels whose yellow contents could be recognized by the naked eye. At times this could be followed for short distances along the bronchi as narrow yellow cords, or when cut transversely appeared as small dots close to the bronchi or vessels. On pressure small droplets of pus may be evacuated, or again where fibrin has led to a coagulation of the exudate a yellow plug can be withdrawn from the channel. These small plugs resembled the thick exudate seen within the bronchi and often were misleading when first viewed. The distribution of the purulent lymphatic masses was most irregular occupying only local or patchy fields in the lung, particularly associated with the purulent confluent pneumonia. In one instance such a lymphatic appeared to be associated with the development of a small abscess lying close to the bronchus. Too much stress cannot be placed upon the importance of the lymphatics in all forms of pneumonia. They play an important role in the drainage of the lung during inflammation. In the normal lung we hardly appreciate the lymphatic distribution except in our observations upon anthracosis. But even under these conditions when much carbon is deposited in conjunction with the lymphatic system we do not gain a true appreciation of the activity of the lymph channels and nodes during an acute process. Bacteria may be demonstrated in acute infections of the lung within the fluid and cells of the lymph channels. Less easily may we demonstrate bacteria in the lymph nodes under similar conditions, although when abscess has occurred their presence is readily recognized. The transport of bacteria is accomplished not only by a passive migration of micro-organisms in the fluid as it drains from the lung, but organisms are also found within the leucocytes as they travel with the current. Only occasionally have we demonstrated bacteria within the wandering large mononuclear cells, although we have observed them in a few instances within the cells lining the sinuses of the nodes. Whether the inflammation of the pleura is directly related to the involvement of the pleural lymphatics we have not been able to determine. In our series of cases pleurisy has not been a prominent feature of the disease, and in many instances the grade of involvement was so slight that it was not easily recognized by the naked eye and showed only a slight reaction microscopically. That the presence of bacteria within the intricate plexus of lymphatics beneath the pleura may be responsible for the development of an inflammation of this membrane may well be the case, and in this way simulate the mode of transmission of the infection as seen in lobar pneumococcus pneumonia and in the streptococcus type of infection. _Abdominal Viscera_