To counteract fermentation, antiseptics (salol, naphthol, freshly burned charcoal) may be given with each meal, along with pepsin and hydrochloric acid. RUPTURE OF THE STOMACH IN SOLIPEDS. Mainly in solipeds. Causes: overloading, fermentation, impossibility of eructation, violent concussions, falls, galop, concretions, dilatation, catarrh, ulcers, cicatrices, abscesses. Symptoms: Anamnesis, colic relieved, followed by prostration, sinking, complete anorexia, tender abdomen, vomiting, no abdominal rumbling. Lesions: tear in greater curvature, most extensive in outer coats, shreddy, bloody edges with clots, contents in omentum, other seats, partial ruptures. Treatment: in partial ruptures, stomach pump, diet. Prevention. This is pre-eminently a disease of solipeds for the reason that they alone of domestic animals are especially liable to overload the comparatively small stomach and are mostly unable to relieve the overloaded viscus by eructation or vomiting. _Causes._ These are in the main overloading of the stomach and overdistension, by the gases of indigestion. To this are usually added violent concussions when the animal throws itself down violently. The stomach distended to the fullest possible capacity, and lodged in a cavity which is not all equally tense, is comparable to a very tense bladder which is liable to burst when forcibly struck, or suddenly compressed. Apart from such indigestion, cases are recorded in which the full stomach has been burst by a sudden fall in the shafts or elsewhere. Miles even records a case which occurred during a rapid galop after a full drink of water. The presence of solid bodies (calculi, gravel) in the stomach or even in the intestines has appeared to cause rupture by blocking the outlet of ingesta and determining indigestion. Certain conditions predispose to rupture, notably dilatation of the stomach with attenuation of its walls, cribbiting, old standing catarrh of the viscus, pre-existing ulcerations, cicatrices and abscesses. _Symptoms._ There is usually the history of a full feed of grain, followed by violent colic, and indications of gastric overdistension, tense abdomen, dullness, then the rejection of the gastric contents by vomiting, the matters escaping by the nose, and then collapse. The violence of the colics may cease, but the pulse becomes rapid, small, and finally imperceptible, the breathing hurried, the head depressed, eyelids, ears and often the lower lips drooping, the face becomes heavy and expressionless, the belly distended and tender, the skin covered with cold sweat, and the temperature exalted above or depressed below the normal. There is never any disposition to eat nor drink. Death follows in a few hours.