Definition. Causes: traumas, poke, stanchions, collar, yoke, interfering, neurectomy, fractures, tumors, callus, rheumatism, gout, violent overdistension. Lesions: nerve sheath red, swollen, exudate, leucocytes in excess, fibroid thickening, nerve atrophy, degeneration, axis granular, myelin in oily globules, peripheral extension. Muscular degeneration and atrophy. Symptoms: tenderness, swelling, muscular atony, wasting, spasms, twitching, decreased excitability, paralysis, in section swelling on proximal end. Prognosis: disability for weeks, months or year; response to electric current, operability of tumors, curability of rheumatism or gout, hopeful conditions; long standing degeneration, etc., unpromising. Treatment: rest, soothe, anodynes, splint with soft pad, essential oils, lead and opium lotion, ice, snow; derivatives; laxatives; diuretics, anti-rheumatics, Faradisation. _Definition._ Inflammation of a nerve leading to paralysis of the parts to which it is distributed. _Causes._ Traumatism is the most common factor. Among the common examples are injury of the seventh nerve above the angle of the lower jaw, by a poke worn in pasture by the horse, or by stanchions in the cow. Hogs may suffer from blows of the triangular neck gear worn to prevent them from breaking through fences. Blows by the yoke, incised and contused wounds implicating the nerve, such as neurectomy, and the blows received in interfering, and compression by tumors or bony growths, are familiar examples. Fractures with displacement, notably those of the sacrum and proximal end of the coccyx with caudal paralysis, are not uncommon. In fractures of the limbs the pressure upon or wounding of a nerve. Again, the callus on the seat of fracture may induce neuritis by pressure, as may also the projection of the end of a bone in luxation. Rheumatism affecting the nerve sheaths and, in birds and swine, gout, are additional factors. Violent overdistension, and even chronic muscular spasm, are quoted as causes. _Lesions._ The early changes are mainly in the connective tissue sheath, which becomes hyperæmic, red and swollen, with a gelatinoid exudate and a great multiplication of leucocytes. Later, the interfibrillar connective tissue is involved and the nervous substance proper undergoes hyperæmia and degeneration. The axis cylinder undergoes granular degeneration and the myelin breaks up into oil-like globules. The lesions are at first limited in extent, though there may be more than one focus, and the resulting degeneration of the nervous filaments advances toward the periphery in accordance with Waller’s law by which disease changes proceed rapidly in parts cut off from their trophic cells.