After a brief mental struggle, she quietly selected the necessary instruments, and studied her criminal design to its smallest details, taking care to avoid discovery. After having formed her plans, she passed the night in quiet sleep, and on the following day committed the deed. In appearance everything was the work of premeditation and clear consciousness. After the deed she astutely made insinuations against her father, who was entirely innocent of complicity; on her knees, by her mother’s body, she declared her own innocence. She carried simulation to a fine point of art, displaying during these days an energy and resolution astonishing in a person so weak. It is clear that her deed had for the time raised her above herself. She had a strange avidity for her mother’s goods. Her great desire was separation from the paternal house and an independent position. After the deed she said that she was no longer in the hands of Satan. In prison she lived for more than three years without giving any sign of mental or of physical disease. She bore herself in an unchanging, composed manner, depressed, free from all eccentricity; it was a consolation to her to know that her father and her sister had forgiven her. At the end of 1886 appeared signs of rapid tuberculosis, to which she succumbed. She died penitent, feeling sure of reconciliation with God. At the autopsy advanced tuberculosis was found in both lungs, also in the kidneys; this was the cause of death. The brain could not be examined immediately, and was therefore preserved. The dura mater, adherent to the cranium externally, was white and lacking in lustre; internally there were bright spots with red maculæ as distinct as in hemorrhagic pachymeningitis. The brain was soft, humid, and very anæmic. Its weight, after the serum in the cavities had flowed away, was 1164 grammes. The occipital lobes did not entirely cover the cerebellum. The form of the brain was elliptic. The sulci appeared deep and large. The parietal and temporal lobes were very large, with great development of the convolutions and numerous atypic clefts. The frontal lobe was small compared to the parietal, and its convolutions compressed. The frontal and occipital convolutions were not atypic except by their slight development. There was scanty development of the frontal and occipital lobes, especially on the left side. _Conclusions._--We have here a real atrophy of the cerebral cortex, which has the characters of a congenital hereditary degeneration. This atrophy is manifested in the insufficient development of the frontal and, still more, the occipital convolutions, in the smallness of the convolutions, in the incomplete covering of the cerebellum by the cerebrum, and by the number of atypical segmentations in the cerebral cortex, representing (at all events in the opinion of Benedikt) a true aplasia.