General Anatomy, Applied to Physiology and Medicine, Vol. 3 (of 3)Bichat, Xavier
Science
General Anatomy, Applied to Physiology and Medicine, Vol. 3 (of 3)
Bichat, Xavier
Human anatomy; Human physiology
In the womb there are two causes of distension; 1st, the sinuses
greatly developed; 2d, an addition of substance, a real momentary
increase of the fibres of the organ which remains as thick and even
more so than in the natural state. At the time of accouchement, the
sinuses immediately flatten by the contraction of the fibres; hence
the sudden contraction of the organ. But as on the one hand nutrition
alone can remove by decomposition the substances added to the fibres to
enlarge them, and as on the other, this function is exerted slowly,
after the womb has undergone the sudden contraction owing to the
flattening of its sinuses, it returns but gradually and at the end of
some time to its ordinary size. Extensibility is not then brought into
action in the womb filled by the fœtus, and in the aneurismatic heart;
these organs really become at that time the seat of a more active
nutrition; they grow preternaturally, as they have grown naturally
with the other organs; but these do not then experience an analogous
phenomenon, they become monstrous in comparison. The womb decreases,
because the motion of decomposition naturally predominates over that of
composition after accouchement, whilst it was the reverse before this
period. The aneurismatic heart remains always so.
These dilatations of the heart should be carefully distinguished from
those really produced by extensibility, as in the right auricle and
ventricle for example, which are found full of blood at the moment
of death, because the lungs which are weakened, not allowing it to
pass through them, compel it to flow back to the place from which it
came. There are but few hearts which do not exhibit in very various
degrees, these dilatations, which we have the power in a living
animal of increasing or diminishing at will, according to the kind of
death we produce. Two hearts are hardly ever of the same size after
death; many varieties are met with, and these depend more or less on
the difficulties which the blood experiences in the last moments,
in passing through the lungs. Hence why in the diseases of the
heart, there is no standard by which we can compare the morbid size,
especially if we examine the organ as a whole. In fact the distension
of the right side can give it an aneurismatic appearance, and a size
even greater than that of some aneurisms. If we examine the left side
separately, the error is more easily proved, because this side is
subject to less variations. But the principal difference consists
in the thickness. The power of contraction appears to increase in
proportion to this thickness, which arises from the substance added by
nutrition. It is this power which produces the great beating that is
felt under the ribs, the strength of the pulse, &c.
_Contractility._
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