Leprosy : $b in its clinical & pathological aspects — John Shaqi
Leprosy : $b in its clinical & pathological aspectsHansen, G. Armauer (Gerhard Armauer)
Science
Leprosy : $b in its clinical & pathological aspects
Hansen, G. Armauer (Gerhard Armauer)
Leprosy
There is, therefore, no other course open to us but to assume the
infectiousness of the disease, and thus the spread of leprosy
is readily understood; while by the assumption of heredity, or
Baumgarten’s latent germinative infection, it remains absolutely
inexplicable. These two last causes are to our thinking absolutely
proved to be non-existent by the case of Father Damien; by the results
of our investigations in North America; and by the diminution of the
disease in the descendants of lepers in the Norwegian towns.
But _direct proofs_ of its contagiousness may also be obtained. Such
are given by Drognat-Landré in his book, and also by many other
observers. Norway can supply many proofs; but against these it can
always be urged that it is impossible to exclude the possibility of
inheritance on account of the widespread nature of the disease. But we
think we can supply from Norway a still better proof in the shape of
the gradual diminution of the disease during the last thirty-five to
forty years.
Up to 1856 leprosy probably increased in Norway; we cannot speak more
definitely, as previous to that year there was no exact or sufficient
enumeration of the lepers. But we have grounds for believing that we
have obtained, by means of the yearly census begun in 1856, a pretty
exact knowledge of the number of new cases in the years 1851-55,
and this shows that the number is considerable, and almost exactly
corresponds with the number of new cases in the next five years,
1856-60. This, we think, indicates that the total number of lepers in
the two quinquenniums was pretty much the same. Now we know that there
were in Norway in 1856 over 2800 lepers. The number was estimated
in 1836 at 659, and in 1845 at 1122. According to this estimate a
considerable increase had taken place. We can, however, with certainty
say that the numbers for 1836 and 1845 are too low. In the first place,
the enumeration was not undertaken by medical men, and secondly, even
now, there are many lepers overlooked at every enumeration, or, to
speak more correctly, they are not discovered because the patients
conceal their condition. But that the number of those overlooked in
the years 1836 and 1845 can have been so great as the difference
between the numbers for those years and that for 1856 is scarcely
credible, and it therefore appears probable that the number of lepers
in Norway increased during the first half of the century.
In the year 1856 the first Leper Asylum was opened in Bergen.
Previously to this we had in Bergen the St. Georg and Lungegaard’s
Hospitals, which together served for 200 patients. In Molde there was
also a small hospital, for lepers, so that altogether in the year 1856
there were about 235 lepers in hospital. In 1861 were opened two new
asylums, one in Molde, the other in Trondhjem (Drontheim), and as is
seen in TABLE IV (p. 145), since that time a large number of lepers
have been admitted to these institutions.
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