Mental diseases: a public health problemMay, James Vance
Science
Mental diseases: a public health problem
May, James Vance
Mental health services; Psychology, Pathological
Temporal Lobe—Lesions in the posterior half of the first temporal may
cause Wernicke's sensory aphasia and a subcortical involvement, word
deafness. Bilateral destruction of the first and transverse temporals
causes cortical deafness. Extensive bilateral lesions in the lower
part of these lobes result in mind deafness. Irritative lesions in the
uncinate gyrus lead to hallucinations of taste and smell, with smacking
of the lips and tongue movements.
Island of Reil—Lesions of the anterior part cause symptoms resembling
Broca's motor aphasia. Lesions of the posterior part result in symptoms
suggesting Wernicke's sensory aphasia. Transcortical motor and sensory
aphasia may result.
Occipital Lobes—Lesions of the calcarine area give rise to
hemianopsia, and bicortical involvements lead to cortical blindness.
Bilateral lesions of the lateral surface may cause mind blindness.
Disturbances in the centrum ovale may cause monoplegias or
monoanesthesias, and lesions in the corpus callosum, apraxic symptoms.
Characteristic of cerebellar lesions are ataxias and disturbances of
equilibrium, often with vertigo and paroxysmal vomiting.
An involvement of the corpora quadrigemina may cause pupillary changes,
unilateral or bilateral paralysis of eye muscles, nystagmus, visual
disturbances, deafness and ataxia or anesthesia.
Lesions of the cerebral peduncles may give rise to very characteristic
syndromes. If the tegmentum and pes pedunculi (basis pedunculi) are
both involved, there may be a complete hemiplegia of the opposite side
with an oculomotor paralysis on the same side (Weber-Gubler syndrome).
Or there may be in addition to this a marked tremor in the limbs of the
paralyzed side (Benedikt's syndrome). A unilateral oculomotor paralysis
may be combined with a cerebellar ataxia (Nothnagel's syndrome).
The thalmic syndrome of Déjerine and Roussy shows a contralateral
hemianesthesia, violent and persistent pains on the anesthetic side,
hemiataxia, hemichorea or hemiathetosis, slight temporary hemiparesis
and sometimes hyperesthesia. Lesions further back, possibly involving
the internal capsule, may cause hemianesthesia of touch, pain and
temperature senses.
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