Some years ago the late Dr. Van Fleet made numerous crosses between the Japanese and the American chestnuts, the Chinquapin, and other species and varieties. Personally, I have not been in very close touch with Dr. Van Fleet's experiments. Doubtless some of you know more about them than I do. Regarding these I will only say at this time that the work begun by Dr. Van Fleet is being continued by the Federal Bureau of Plant Industry, with Mr. G. F. Gravatt in direct charge of the work so far as the Office of Investigations in Forest Pathology is concerned. Mr. Gravatt is also testing out the value of scions taken from seemingly resistant native trees when grafted on resistant stocks. Some years after the blight had destroyed most of the chestnut trees in the northeastern states we kept getting reports from various localities to the effect that the blight was apparently dying out. Many of these reports came from sources that made us doubt their value, but others came from more reliable sources. We have had opportunity to investigate a number of these reports and have usually found that the statement that the blight was dying out was, in a sense, strictly true, the reason being that the chestnut trees were entirely dead, except for sprouts. This fact naturally prevented the disease from showing us as much as in former years. Some twelve years ago I noticed in Pennsylvania a sprout of an American chestnut about an inch in diameter which had a typical hypertrophy of the disease, apparently completely girdling the sprout at its base; also a girdling lesion farther up on the stem. The hypertrophy was such a pronounced one and in other respects such a typical example of the disease that I photographed it. A few years later I was surprised to observe that this sprout had increased to more than three times its former diameter and that the two diseased areas just mentioned apparently had disappeared--at least they were no longer in evidence except as rough-barked areas. To make a long story short this sprout is still alive and has increased in size and height each year. Although now (1924) it is considerably branched and makes a small bushy tree it is badly diseased in numerous places and is only partially alive, but the dead portions have not resulted from some half dozen of the original disease lesions (apparently girdles), but from later infections. The very fact that a sprout should have lived for more than twelve years in the center of one of the most badly diseased areas known to the writer seems at least to suggest the possibility that future chestnut sprouts may yet grow in spite of the disease and persist--at least in a shrubbery form if not as a tree.