Shell-shock and other neuropsychiatric problems : $b Presented in five hundred and eighty-nine case histories from the war literature, 1914-1918Southard, Elmer Ernest
History
Shell-shock and other neuropsychiatric problems : $b Presented in five hundred and eighty-nine case histories from the war literature, 1914-1918
Southard, Elmer Ernest
War neuroses; World War, 1914-1918 -- Medical care; World War, 1914-1918 -- Psychological aspects
At autopsy the spine and dura mater proved normal; but microscopically
serial sections through the fourth and fifth dorsal segments showed
softening of the right anterior horn and posterior columns, with
cavitation in the radicular zones, and the white matter of the fifth
dorsal segment was in a state of acute degeneration. There were also
ependymal changes, namely, at the fifth dorsal level a dilatation with
deposit of albumin; in the lumbar region, breakage of the ependymal
wall, with cellular gliosis. The dilated ependyma was surrounded by
an area of fibrillary gliosis which had proliferated in the form
of a septum in the interior of the canal. (According to Claude and
Lhermitte, these data concerning hydromyelia, which they regard as
secondary to trauma, are an argument in favor of the traumatic origin
of certain syringomyelias. They regard the breakage of the ependymal
wall as due to hypertension of the spinal fluid due to mechanical
lesions.) Their interpretation of such acute degeneration as was found
in the fifth segment is that this degeneration, as well as that of the
posterior roots, is due to the direct impact of the cerebrospinal fluid
upon the cord structure. As for the softenings with cavitation, they
regard them as surely due to spinal concussion and as very possibly due
to an ischemic necrosis, suggesting that older work by Duret and Michel
on concussion of the brain indicates the possibility of a temporary
ischemia of the spinal cord from the violent impact of the spinal
fluid upon the cord due to shock of the spinal column. The transient
hypertension of the spinal fluid might well induce, they believe, a
vascular spasm with anemia, to which the gray matter is well known to
be especially sensitive. In the present case, a period of somewhat less
than six weeks had sufficed to produce secondary degenerations above
and below the fifth dorsal segment, of a quite classical sort.
Accordingly, we here deal with a severe form of spinal concussion due
to a shellburst, in which intraspinal lesions were produced without
spinal fracture or penetration either of bone or of shell fragments
into the spinal cord or the spinal fluid.
Shell explosion (1 meter distant) kills a soldier by bursting both
lungs within the intact thoracic cage.
=Case 201.= (SENCERT, January, 1915.)
Public-domain text, read in full here on John Shaqi.
Reviews
Reviews
No reviews yet
Be the first to share your thoughts on this work.
Elsewhere in the archive
Join the Discussion
Join the discussion
Sign in to leave a comment or review.
Sign InorCreate an account