Text book of veterinary medicine, Volume 2 (of 5)Law, James
Science
Text book of veterinary medicine, Volume 2 (of 5)
Law, James
Veterinary medicine
_Lesions._ As these are seen only in fatal cases, the presence of an
abscess is the characteristic feature. This is usually submucous, or
less frequently subperitoneal, and may vary in size from a hazelnut
upward. The tendency appears to be to open into the stomach, though it
may burst into the peritoneum and cause general infection of that
membrane. In case of parasites, the spiroptera or œstrus larva may be
found in the abcess cavity having a narrow opening into the stomach. In
certain cases the abscess on the pyloric sac has been found opening into
the duodenum. Congestion, thickening, puckering into rugæ and laceration
of the adjacent mucosæ may be a marked feature, a circumscribed
catarrhal gastritis complicating the local phlegmon.
_Treatment._ This is less hopeful than in catarrhal gastritis, but
should be conducted along the same lines. The same careful diet, with
daily antiseptics and bitters may prove valuable in limiting the
inevitable suppuration, and, if the pus should escape into the stomach,
in healing the lesion. Sulphites of soda, sulphide of calcium,
chamomile, and quinia, are to be commended and pepsin may be added to
secure at once proteid digestion and antisepsis. Laxatives may be
required to counteract constipation or expel irritants, and these may be
combined with the antiseptics already named or with salol, eucalyptol,
sodium salicylate or other non-poisonous agent of this class.
TOXIC GASTRITIS IN SOLIPEDS.
Causes: caustics or irritants acting on mucosa, accidently, or
maliciously. Symptoms: colics, pinched face, small rapid pulse,
hurried breathing, hyperthermia, sometimes salivation, color of buccal
mucosa, odor, congestion of tongue, thirst, urination, icterus,
albuminuria, analysis of urine or vomited matter. Lesions: congestion,
corrosion, necrosis or ulceration of gastric mucosa, discoloration.
Treatment: antidote, stomach pump, demulcents, coagulants.
_Causes._ Toxic gastritis in solipeds is peculiar in this that it must
be due to one or other of the more caustic or irritant agents, which act
chemically on the tissues, while those agents that require to be
absorbed to establish a physiological irritation are comparatively
harmless. This depends on the fact that few or none of the poisonous
agents are absorbed by the gastric mucosa of the soliped, and if
ingested they must pass on into the duodenum before they can be absorbed
into the tissues and blood-vessels. Hence the horse is injured mainly by
actual caustics like mercuric chloride, zinc chloride, ferric or cupric
sulphate, the caustic alkalies or earths, or alkaline carbonates, and
the mineral acids. These may be taken accidently or administered
maliciously, or as medicines.
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