14. Very probably adult persons frequently acquire intestinal tuberculosis through food containing tubercle bacilli, if the epithelial covering of the intestinal mucosa is defective, or if perhaps there exist ulcers which extend down to the parenchyma of the wall of the alimentary tract. (Exanthematic diseases, typhoid, dysentery, carcinoma, etc.) 15. Whether adult persons in whom the conditions are favorable for an intestinal infection with Tb. will develop primary tubercular lesions in the intestinal wall, or in the mesenteric glands and the peritoneum, will depend mainly on the circumstance whether or not, owing to a previous infection, they have become oversensitive to tuberculin. _Individuals oversensitive to tuberculin are inclined to develop lesions at the point of entry of the tubercular virus_, if opportunity is given for the introduction of the virus by means of leucocytic wandering cells. This opportunity is lacking in the virile infecting period at such places where the lymphatic receptive apparatus is destroyed [verödet]. (Mucous surfaces of the faucial ring of consumptives?) 16. In order to explain the mode of origin of cheesy pneumonias and tubercular broncho-pneumonias it is necessary at autopsy to regard most carefully the possible direct extension of the infection from cheesy mediastinal and bronchial glands to the bronchi and their branches, before thinking of aerogenous or hæmatogenous pathogenesis. (Compare [Ribbert] Sievers, “Marburg Dissertation,” Aug. 14, 1902.) 17. Critical analysis of several statistical statements which seek to show that alveolar pulmonary tuberculosis is referable directly to inhaled tubercle bacilli; especially the statement of Knopf (New York) cited by Mitulescu (_Zeitschrift für Hygiene_, 1903), that in Lansing, Mich., twenty employés of a library became consumptive through handling Tb. laden books.[5] Probability of the correctness of my assumption that Knopf was misled by unscientific communications from Lansing. Proof that Mitulescu again misunderstood Knopf. 18. It has not yet been proved that persons cutaneously infected with human or bovine Tb. have as a result of this developed phthisis. (My own observations on cases of infection on the hand in persons working with tubercle bacilli of various origins.) 19. Justification of the statement by Virchow in his “Phymatie, Tuberculose und Granulie,” that “the history of phthisis is concerned much more with cheesy hepatization than with tubercles” (Virchow’s “Tuberkel,” Begriff). 20. The analysis of the origin of pulmonary consumption must begin with the primary attack (primary regionally as well as chronologically) of the Tb. introduced into the organism.