fibrin-content, few flocculi, and relatively small number of white cells. Inflammatory exudates are turbid, often foul-smelling, usually acid, specific gravity over 1.016, high albumin-, fibrin- and urea-content, numerous thick flocculi and numerous cells. In early peritoneal tuberculosis the fluid may be clear and resemble that of a transudate. Milky and opalescent fluids are found in diabetes, lipæmia, new-growths of the peritoneum, obstruction or rupture of thoracic duct or receptaculum. Hemorrhagic exudates may be traumatic (rupture of spleen, liver, intestines, extra-uterine pregnancy, etc.), inflammatory (severe acute peritonitis), or due to new-growths or tuberculosis of the peritoneum, extreme portal stasis, perforation of gastric or typhoid ulcers, severe intoxications, chronic icterus, etc. Red effusions may be due to diffused hæmoglobin. In such cases there is no settling of the color, and coagulation may not occur. When red cells are present settling takes place on standing. Rupture of gall-bladder or bile-ducts may lead to presence of free bile in the peritoneal cavity. Postmortem diffusion of bile through the gall-bladder wall should not be mistaken for a pathologic condition. In normal conditions there is just enough fluid in the peritoneal fluid to make the surfaces moist, and about a teaspoonful in all may be collected from the flanks and pelvis. The amount may be greatly increased just before death in all cases of slowly progressive cardiac weakness. Note character of peritoneum (normally moist-shining, grayish, translucent, cloudy, dry, lustreless, thickened, hyaline (“iced” or “Zuckerguss”) in chronic inflammation.) 4. =Omentum.= Note position of lower border, amount of fat, condition of blood-vessels, dry or moist-shining surface, adhesions (to appendix, cæcum, oviducts), indurations, contractions (edges rolled up), character of lymphnodes, cysts, tubercles, secondary tumors, snared-off tumors from ovary or uterus (parasitic cysts, fibroids), encysted foreign bodies, etc., exudates on surface, fat-necrosis, accessory spleens, encysted parasites, hernia, etc. Most common pathologic conditions are inflammation (secondary to appendicitis, salpingitis, etc.), metastic carcinoma and tuberculosis.